3 Primary Organs to Fix in Type 2 Diabetes
People who are an overweight and sedentary lifestyle...they are in higher % risk of Type 2 diabetes (T2DM).
However, NOT everyone become T2DM, but what is different between a person who has T2DM and not T2DM...
They are some risks that you can not change such as family health history, age, and ethnicity.
However, there is something you can change such as lifestyle, habits, and physical activity and so it is preventable to be T2DM.
so what do we need to focus on deeply to prevent T2DM??
First, let's focus on the characteristic of T2DM.
The pancreas secretes insulin, but not enough.
The liver can not detect blood glucose level
The liver does not notice the insulin (insulin resistance)
The liver does not detect fat
Your muscle cannot detect blood glucose (insulin resistance)
not enough muscle mass (no storage of energy)
So that you have more glucose and fat are floating around in your blood which makes a microscopic scar on your vein, and eventually, it cause of cardiovascular problems, high blood pressure, blindness, kidney failure, and nervous system disorders.
Now, as you see, Pancreas, Liver, and Muscles are not performed well for people who have Type 2 Diabetes.
Hummmm...so I see, Pancreas, Liver & Muscles are the 3 organs to fix...
Okay, so what we should focus on to fix those performances?
Reduce Triglycerides (fat) from liver and pancreas, and
Gain muscle mass
Research & study found out "for people with Type 2 diabetes, losing weight allows them to drain the excess fat out of the pancreas and allows functions to return to normal"(Science Daily)
Non-alcoholic fatty liver disease (NAFLD) is highly prevalent in type 2 diabetes mellitus (T2DM), likely reflecting the frequent occurrence of obesity and insulin resistance in T2DM.
Liver fat content can be decreased by weight loss and by a low as compared to a high-fat diet. (Järvinen 2015)
In a cross-sectional study, every 10% increase in the ratio of skeletal muscle mass to total body weight was associated with an 11% reduction in risk of insulin resistance and a 12% drop in risk of transitional, prediabetes, or overt diabetes, Preethi Srikanthan, MD, of the University of California Los Angeles, and colleagues reported online in the Journal of Clinical Endocrinology and Metabolism.
The results may also have implications for the role of muscle-building exercises in preventing metabolic dysfunction, Srikanthan and colleagues wrote.